Overweight and obesity
Excess weight is not about willpower. It is a hormonal and metabolic issue. Often the root cause is not “eating too much, moving too little,” but chronically high insulin (the hormone that controls blood sugar), leptin resistance (when the brain stops getting the “I’m full” signal), and a broken satiety signal.
- 70%+ of Estonian adults are overweight or obese
- “Calories in – calories out” ignores hormonal context
- Reversible with the right protocol and patience

What does “overweight” actually mean for your metabolism?
Doctors classify weight using BMI (body mass index – your weight divided by your height squared): 25–29.9 is overweight, 30 or above is obesity. But BMI is a rough tool – it can’t tell muscle from fat, or show where the fat sits on your body. Waist size and visceral fat (fat around your internal organs) matter more for your health.
Body fat is not just storage – it works like an active hormone-producing organ. It releases leptin (the fullness signal), adiponectin (which helps cells respond to insulin), and substances that cause inflammation. When there is too much fat – especially around the organs – it throws off your hormone balance and makes it easier to keep gaining weight.
In most cases, being overweight is a result of insulin resistance and leptin resistance, not the cause. Insulin resistance means your cells stop responding well to insulin (the hormone that manages blood sugar). Leptin resistance means your brain stops receiving the “I’m full” signal. High insulin blocks fat burning, and the fullness signal never reaches the brain. That’s why you feel hungry, tired, and keep gaining weight – no matter how much willpower you have.
Signs it’s not just about eating too much
- Waist size of 94 cm or more in men, 80 cm or more in women
- Weight won’t drop even when eating fewer calories
- Sugar cravings in the evening, hard to stop eating at dinner
- Hunger returns within 2–3 hours of eating
- Feeling tired and drowsy after lunch
- Eating at night or waking up hungry
- Puffiness (face in the morning, tight rings or shoes in the evening)
- Weight comes right back after every diet
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KMI, vööümbermõõt ja kehakoostise normid
Riskihindamine täiskasvanule
| Marker | Optimaalne | Ülekaal | Kõrge risk |
|---|---|---|---|
| BMI (kg/m²) | 18,5–24,9 | 25,0–29,9 | ≥ 30,0 |
| Vööümbermõõt (M) | < 94 cm | 94–102 cm | > 102 cm |
| Vööümbermõõt (N) | < 80 cm | 80–88 cm | > 88 cm |
| Vöö / puusa suhe (M / N) | < 0,90 / 0,85 | 0,90–1,0 / 0,85–0,90 | > 1,0 / > 0,90 |
| Kehakoostis: rasva % (mehed) | 10–20 % | 20–25 % | > 25 % |
| Kehakoostis: rasva % (naised) | 18–28 % | 28–33 % | > 33 % |
| Vistseraalne rasv (skoor) | 1–9 | 10–14 | ≥ 15 |
BMI (kg/m²)
- Optimaalne
- 18,5–24,9
- Ülekaal
- 25,0–29,9
- Kõrge risk
- ≥ 30,0
Vööümbermõõt (M)
- Optimaalne
- < 94 cm
- Ülekaal
- 94–102 cm
- Kõrge risk
- > 102 cm
Vööümbermõõt (N)
- Optimaalne
- < 80 cm
- Ülekaal
- 80–88 cm
- Kõrge risk
- > 88 cm
Vöö / puusa suhe (M / N)
- Optimaalne
- < 0,90 / 0,85
- Ülekaal
- 0,90–1,0 / 0,85–0,90
- Kõrge risk
- > 1,0 / > 0,90
Kehakoostis: rasva % (mehed)
- Optimaalne
- 10–20 %
- Ülekaal
- 20–25 %
- Kõrge risk
- > 25 %
Kehakoostis: rasva % (naised)
- Optimaalne
- 18–28 %
- Ülekaal
- 28–33 %
- Kõrge risk
- > 33 %
Vistseraalne rasv (skoor)
- Optimaalne
- 1–9
- Ülekaal
- 10–14
- Kõrge risk
- ≥ 15
KMI on ligikaudne – lihaselisel inimesel võib see näidata ülekaalu ka siis, kui rasva on vähe. Vööümbermõõt ja kehakoostise analüüs on täpsemad.
Frequently asked questions
Related topics
Metabolic health is interconnected – explore the other deep-dives:
Insulin resistance
The root of metabolic disease – 5–10 years before diabetes appears.
ReadVagus nerve
The rest-and-repair system – digestion, heart, inflammation, stress.
ReadCholesterol & ApoB
Expanded lipid panel – ApoB, Lp(a), homocysteine, A vs B phenotype.
ReadSleep
The foundation of metabolic health – hormones, cortisol, insulin, brain recovery.
ReadChronic inflammation
The ‘silent fire’ behind cardiovascular disease, diabetes and autoimmunity.
ReadLow-carb and keto
Carbohydrate restriction – IR, T2D, fatty liver, metabolic syndrome.
ReadAgeing
Glycation, insulin, inflammation and muscle – what you can influence yourself.
ReadTeaduslikud allikad
- Fothergill E et al. Persistent metabolic adaptation 6 years after “The Biggest Loser” competition. Obesity, 2016. doi.org/10.1002/oby.21538
- Hall KD, Kahan S. Maintenance of Lost Weight and Long-Term Management of Obesity. Medical Clinics of North America, 2018. doi.org/10.1016/j.mcna.2017.08.012
- Frayling TM et al. A common variant in the FTO gene is associated with body mass index and predisposes to obesity. Science, 2007. doi.org/10.1126/science.1141634
- Wilding JPH et al. Once-Weekly Semaglutide in Adults with Overweight or Obesity (STEP 1). NEJM, 2021. doi.org/10.1056/NEJMoa2032183
- Leidy HJ et al. The role of protein in weight loss and maintenance. American Journal of Clinical Nutrition, 2015. doi.org/10.3945/ajcn.114.084038
- Jakubowicz D et al. High caloric intake at breakfast vs. dinner differentially influences weight loss. Obesity, 2013. doi.org/10.1002/oby.20460
- Westerterp-Plantenga MS. Sleep, circadian rhythm and body weight. Proceedings of the Nutrition Society, 2016. doi.org/10.1017/S0029665116000227
- Villareal DT et al. Aerobic or Resistance Exercise, or Both, in Dieting Obese Older Adults. NEJM, 2017. doi.org/10.1056/NEJMoa1616338
Dr Sergey Saadi – expert in metabolic health and the root causes of weight gain
Dr Sergey Saadi is a physician with over 15 years of clinical practice.
- Thousands of consultations for metabolic-health and weight concerns
- Author of “Dangerous secrets of blood sugar” (Varrak)
- Clinical approach: body composition + hormonal panel + lifestyle audit
- No one-size-fits-all diets – individual root-cause analysis
This page is educational and general. It is not personal medical advice, a diagnosis or a treatment plan. Discuss changes to your health, medications or supplements with your doctor.